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Tibet
Священный Кайлас и озеро Манасаровар. 21-25 дней глубокого погружения в буддийскую традицию.
Nepal
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News and Articles
Current materials on health, holistic medicine and my programs
TSH Is a Pituitary Hormone, Not a Thyroid Hormone: The Single-Test Blind Spot
When a clinician orders "just TSH" — they measure pituitary function, not thyroid function. TSH is a late, inertial marker: it reflects chronic imbalance, not acute dysfunction. Estrogens, biotin, non-thyroidal illness, circadian rhythm — all shift the number without changing thyroid status. Here is the full panel and the decision principle.
June 2, 2026T3 — the Only Active Thyroid Hormone: Why It Works Inside the Cell
T3 is the only thyroid hormone that actually works. And it works not in blood but inside the cell, at the nuclear receptor. Blood is only a transport phase. Up to 40% of women with normal TSH have cellular hypothyroidism and suffer with full symptoms despite "normal" labs. Here is the biology of T3, T4→T3 conversion, the hidden forms of deficiency, and the full laboratory panel.
June 2, 2026Hypothyroidism Treatment Beyond Thyroxine: Conversion, Cofactors, Cortisol
The patient has been on levothyroxine for five years, TSH is normal — yet hypothyroid symptoms persist. This is not "poor compliance"; it is a systems failure: peripheral T4→T3 conversion, membrane transport, cofactors, cortisol, gut. We break down what general practice misses and what protocol actually works — with PMIDs, doses, and 8-week control markers.
June 2, 2026Stress and the Thyroid: Three Levels of Suppression and the fT3/rT3 Protocol
TSH normal, free T4 normal, and the patient is falling apart with fatigue, cold intolerance, weight gain, and broken sleep. The classic "your labs are fine" trap. In reality, chronic stress shuts the thyroid down at three different levels, and you only see it on a properly built panel: fT3, rT3, salivary cortisol, ferritin, selenium. Here is the mechanism, the exact numbers, and an 8-week recovery protocol.
June 2, 2026Thyroid After Virus: Three Scenarios — Subacute Thyroiditis, Autoimmune Shift, NTIS
Two to six weeks after an acute viral infection, the patient returns with "I am not myself" complaints: palpitations, sweating, anxiety — or the opposite, fatigue, edema, apathy. The post-viral thyroid can follow one of three clinically distinct paths. Treatment for each is different. Blanket therapy makes things worse. This review covers how to tell the scenarios apart, what to measure, and when not to prescribe levothyroxine.
June 2, 2026Anti-TPO: The Main Marker of Hashimoto's Thyroiditis and How to Interpret It
Anti-TPO antibodies are the key to diagnosing autoimmune thyroiditis — but not a treatment target and not a reason for repeat testing. Here is when the test changes management, how to read the titer (35 / 100 / 1000 IU/mL), why an isolated positive without ultrasound and clinical signs is a lab finding rather than Hashimoto, and where selenium actually reduces antibody levels by 30–40%.
June 2, 2026Hashimoto's Thyroiditis: Four Stages of Disease Evolution
Hashimoto's thyroiditis is not a snapshot diagnosis — it is a slow autoimmune process that unfolds over 10–30 years through four sequential stages. Understanding the stage changes clinical strategy: starting levothyroxine in stage 1 is blind therapy; withholding it in stage 3 is malpractice. This article maps the four stages with markers, timing, and the protocol grounded in ATA Guideline.
June 2, 2026Thyroid Nodules and TIRADS: When Biopsy, When Surveillance
Up to 50% of adults have thyroid nodules on ultrasound. The vast majority are benign. The decision to biopsy is not made on the fact of detection but on the TIRADS category combined with size. This review covers the practical ACR TIRADS scale, size thresholds for biopsy, the surveillance protocol, and where Bethesda fits after FNA.
June 2, 2026Selenium 200 mcg for Hashimoto's — The Precise Clinical Dose
Selenium is the obligate cofactor of deiodinases and glutathione peroxidases, and the thyroid gland concentrates it more densely than any other organ per gram of tissue. In Hashimoto's thyroiditis, 200 mcg for 6 months reduces TPO antibodies by 30–40%. Less is sub-optimal, more is selenosis. This is the analysis: why exactly this dose, which form works, when selenium is useless, and how to monitor therapy.
June 2, 2026TSH Norms in Pregnancy: Trimester Targets and Clinical Tactics
What is "normal" for a non-pregnant woman can be hypothyroidism for the fetus. Until weeks 18–20, the fetal CNS develops entirely on maternal thyroid hormones. Here are trimester-specific TSH targets, tactics for anti-TPO positive women, levothyroxine dosing, and iodine prophylaxis per ATA 2017 (PMID 28056690).
June 2, 2026Vitamin D and K2 in Hashimoto: the biological pair that decides where calcium goes
Vitamin D raises calcium absorption but does not decide where that calcium goes. Vitamin K2 (menaquinone MK-7) does: into bone via osteocalcin activation, or into arterial walls when deficient. In Hashimoto autoimmune thyroiditis this pair is critical — hypothyroidism itself increases the risk of osteoporosis and vascular calcification. Exact doses, 25(OH)D targets, monitoring, contraindications, and the protocol with levothyroxine.
June 2, 2026After Thyroidectomy: L-Thyroxine vs NDT — Clinical Criteria for Choosing
After thyroid removal, 10–15% of patients on adequate L-thyroxine and "normal" TSH continue to report fatigue, weight gain, cognitive issues. This is not "all in your head" — it is incomplete T4-to-T3 conversion. I break down the US protocol for NDT: composition, brands (Armour, NP Thyroid, ADTHYZA), starting doses, titration, and when L-thyroxine remains the correct choice.
June 2, 2026Ready to start your health journey?
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